Large-Scale Study Proves High-Fat Diet Promotes Health and Longevity

  • High intakes of healthy fats – especially saturated fats – boost health and longevity. Those who got 35 percent of daily calories from healthy fats were 23 percent less likely to die than those who only got 10 percent of calories from fat.
  • People who got 77 percent of daily calories from carbohydrates were 28 percent more likely to die than those who got 46 percent of calories from carbs.
  • Other recent research shows a reduced-sugar diet can lower liver fat by more than 20 percent in just nine days.
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Mitochondria – the tiny energy factories within your cells – generate adenosine triphosphate (ATP), the energy currency your body needs to run its systems. Your mitochondria are also responsible for apoptosis (programmed cell death), and serve as important signaling molecules that help regulate your genetic expression. Hence, the state of your mitochondria plays a key role in health and disease.

Once your mitochondria become damaged and dysfunctional, your energy reserves decrease, leading to a wide variety of symptoms, some of the most common being headache and fatigue, and leaving you increasingly vulnerable to degenerative diseases such as cancer, heart disease, diabetes and neurodegenerative decay.

Unfortunately, mitochondrial damage is more the norm than the exception these days, thanks to the prevalence of processed food diets, inactivity, lack of sun exposure and excessive exposure to toxins and non-native electromagnetic fields from cell phones, routers, cellular towers and more. All of these factors contribute to mitochondrial dysfunction. Fortunately, your body can regenerate and renew, regardless of your age – provided it has the proper fuel to do so.

A ketogenic diet – which is very low in net carbohydrates and high in healthy fats – is key for boosting mitochondrial function.

When your body is able to burn fat for fuel, your liver creates water-soluble fats called ketones that burn far more efficiently than carbs, thereby creating fewer reactive oxygen species (ROS) and secondary free radicals. Ketones also decrease inflammation, improve glucose metabolism and aid the building of muscle mass.

International Study Confirms Fat for Fuel Premise

 The large-scale international study (known as the international Prospective Urban Rural Epidemiology, or PURE, study) reports thathigh intakes of healthy fats – especially saturated fats – boost health and longevity.

Its research team recorded the eating habits of 135,000 adults in 18 countries – and followed the participants’ health for more than seven years on average. Those with the highest intake of dietary fat (35 percent of daily calories) were 23 percent less likely to have diedduring the study period than those with the lowest fat intake (10 percent of calories).

The rates of various cardiovascular diseases were essentially the same across fat intake, while strokes were less common among those with a high fat intake. The participants with the highest carbohydrate intake (77 percent of daily calories) were 28 percent more likely to have diedthan those with the lowest carbohydrate intake (46 percent of calories).”

Low-Fructose Diet Significantly Reduces Liver Fat in Mere Days

 Another recent study found a reduced-sugar diet lowered liver fat by more than 20 percent in just nine days. Processed fructose found in soda, fruit juices and processed food is a major contributor to nonalcoholic fatty liver disease (NAFLD).

The study clearly showed that sugar is turned into fat, which may explain the epidemic of fatty liver in children consuming soda and food with added sugar. The study found that fatty liver is reversed by removing added fructose from our diet.

NAFLD raises your risk for Type 2 diabetes. A high-fat, low-carb diet has been shown to improve both blood sugar levels and blood lipids.

Dr. Robert Lustig (who investigated the role of fructose in disease for many years) commented on the results, saying, “Many people think that fructose provides empty calories. But no, they are toxic calories because they are metabolized onlyin the liver, and the liver turns the excess into fat.”

American Heart Association Has It All Wrong

In June 2017, the American Heart Association (AHA) shocked health conscious individuals around the world by declaring coconut oil dangerous.

However, the AHA had carefully cherry-picked outdated data from the 1960s and early 1970s. Moreover, none of these four studies actually involved coconut oil, which means the AHA is flat out making false claims when it’s identifying coconut oil as a dangerous fat.

Nutritional science has made significant strides since the ’70s, and many recent studies have repeatedly refuted the idea that high-fat diets promote heart disease.

Why Is the AHA Clinging to Outdated Science?

As noted by Nina Teicholz, an investigative journalist and author of “The Big Fat Surprise: Why Butter, Meat, and Cheese Belong in a Healthy Diet:”

“To me, the AHA advisory released in June (2017) was mystifying. How could its scientists examine the same studies as I had, yet double down on an anti-saturated fat position? With a cardiologist, I went through the nuts and bolts of the AHA paper, and came to this conclusion: It was likely driven less by sound science than by longstanding bias, commercial interests and the AHA’s need to reaffirm nearly 70 years of its ‘heart healthy’ advice …

Why the AHA is resistant to updating its view of saturated fats, despite so much legitimate science, could simply reflect the association’s unwavering devotion to a belief it has promoted for decades. Or it could be due to its significant, longstanding reliance on funding from interested industries, such as the vegetable-oil manufacturer Procter & Gamble, maker of Crisco …

In July of 2017, Bayer, the owner of LibertyLink soybeans, pledged up to $500,000 to the AHA, perhaps encouraged by the group’s continued support of soybean oil, by far the dominant ingredient in the ‘vegetable oil’ consumed in America today.”

Consider Experimenting With a Water Fast

A Multi-day fasts is where the only thing you consume is water and mineral supplements, and no other food or drink.  It allows your body to seriously upregulate autophagy (destruction of damaged cells) and removes most of the damaged and premalignant cells in your body. It is a magnificent way to help cancer-proof your body. It is also outstanding for helping you achieve optimal body weight, and it can improve your health and extend your lifespan.

The more comfortable way to start a water-only fast is to fast 12 to 18-hour daily intermittent fasts for a few months. This will radically decrease any negative side effects. Your doctor should monitor fasts longer than 18 hours.

During his fast, Dr. Mercola experienced no hunger pains, no food cravings, lost 10 pounds. His ketones were 5.1, and his was blood sugar 45, which doubled the ketone/glucose index threshold Dr. Thomas Seyfried (a leading researcher) says is necessary to treat cancer.

Read more about the water-only fast in Dr. Mercola’s book, Fat For Fuel. Here is a link https://www.fatforfuel.org/

The Benefits of a Cyclical Ketogenic Diet

The cyclical or targeted ketogenic diet is high in healthy fats and fiber, low in net carbs with a moderate amount of protein.

Ideally, once you have established ketosis you cycle healthy carbs back in to about 100 to 150 grams on the few days a week you do strength training.

Health Benefits:

Weight loss – By rebalancing your body’s chemistry, weight loss and/or improved weight management becomes nearly effortless. Studies have shown a ketogenic diet can double the weight lost compared to a low-fat diet.

Reduced inflammation – When burned for fuel, dietary fat releases far fewer ROS and secondary free radicals than sugar. Ketones are also very effective histone deacetylase (HDAC) inhibitors that effectively reduce inflammatory responses.

Reduced cancer risk – While all cells can use glucose for fuel, cancer cells lack the metabolic flexibility to use ketones. Regular cells thrive on them. Once your body enters a state of nutritional ketosis, cancer cells are more susceptible to being removed by your body through a process called autophagy. A cyclical ketogenic diet is a fundamental, essential tool that needs to be integrated in the management of nearly every cancer.

Increased muscle mass – Ketones spare branched-chain amino acids, thereby promoting muscle mass. However, make sure to implement cyclic ketosis. Chronic ketosis may eventually result in muscle loss as your body is impairing the mTOR pathway, which is important for anabolic growth.

Lowered insulin levels – Keeping your insulin level low helps prevent insulin resistance, Type 2 diabetes and related diseases. Research has demonstrated that diabetics who eat a low-carb ketogenic diet are able to significantly reduce their dependency on diabetes medication and may even reverse the condition.

Lowering insulin resistance will also reduce your risk of Alzheimer’s. Recent research strengthens the link between insulin resistance and dementia even further, particularly among those with existing heart disease.

Mental clarity – One of the first things people really notice once they start burning fat for fuel is that any former “brain fog” lifts, and they can suddenly think very clearly. Ketones are a preferred fuel for your brain; hence, the improved mental clarity.

Increased longevity

One of the reasons you can survive a long time without food is due to the process of ketosis, which spares protein breakdown. A fairly consistent effect seen in people on a ketogenic diet is that blood levels of leucine and other important structural proteins go up, allowing these proteins to perform a number of important signaling functions.

Ketones also mimic the life span extending properties of calorie restriction (fasting), which includes improved glucose metabolism; reduced inflammation; clearing out malfunctioning immune cells; reduced IGF-1, one of the factors that regulate growth pathways and growth genes and is a major player in accelerated aging; cellular/intracellular regeneration and rejuvenation (autophagy and mitophagy).

Take Control of Your Health – And Help Others Do the Same

Excessive net carbohydrate intake is the No. 1 culprit behind skyrocketing obesity, diabetes and chronic disease rates, primarily by decimating your mitochondrial function.

Take control – eat a diet that allows your body to burn fat as its primary fuel rather than sugars. Weight loss is simply a natural side effect of rebalancing your body’s chemistry.

This Post has been condensed from Dr. Mercola’s post:https://articles.mercola.com/sites/articles/archive/2017/09/11/ketogenic-promotes-health-longevity.aspx?fbclid=IwAR1hZ3AAi61X9FLBmnBahOLaGCGAq2F3a6zMrEvyhvwzgjwEfJzFZpaHSf8

Please see the original for the Footnotes and Citations for the scientific studies.

I invite you to Follow my Blog, Facebook or be added to my email distribution list. My focus is to maximize my physical performance and mental clarity, body composition, and most importantly overall health with a wholesome diet and exercise.

I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

 To follow my Blog, please click the Follow button to receive an email when the next posting is available. Hint: You may have to click the Accept and Close button before follow is available.

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May you Live Long Healthy.

Yours truly,

Lydia Polstra

Email: lpolstra@bell.net

Facebook: https://www.facebook.com/2healthyhabits/

Blog: https://2healthyhabits.wordpress.com

Disclaimer: The content of this email or Post is not intended for the treatment or prevention of disease, nor as a substitute for medical treatment, nor as an alternative to medical advice. Use of recommendations is at the choice and risk of the reader.

The Ketogenic Diet Starves Cancer Cells.

Dr. Mercola interviews Travis Christofferson, the author of Tripping over the Truth: How the Metabolic Theory of Cancer Is Overturning One of Medicine’s Most Entrenched Paradigms

Christofferson:  Drugs targeting DNA have been very, very disappointing. We’ve been told that these targeted therapies were going to result in cures. It’s clear that’s not the case because of the data from the Cancer Genome Atlas project. There’s just too much random diversity within the genes. It’s hard enough to find a target from one patient to the next and if you do there’s another phenomenon called intra heterogeneity, which is the difference in mutations from cell to cell within the same tumor.

James Watson Nobel, who won the Nobel Prize in 1954 for discovering the structure of DNA, noticed this and said; I am no longer giving money to the Cancer Genome Atlas project. If we’re ever going to cure cancer we’re clearly going to have to go back to the days of Warburg and focus on the metabolism.

Mercola: Warburg was the first person who noted that there’s a difference in the metabolism. Cancer cells are anaerobically metabolizing glucose (without oxygen) inefficiently and they have this relative primitive form of energy generation and it’s inefficient.

So the question becomes what caused them to revert to this? Peter Peterson out of Hopkins took it to the next step, he actually determined that there is a radically reduced number of mitochondria. There are typically several thousand in each cell comprising about a third to 50% of the volume of the inside of your cell. These generate the energies of our cells.

If you’ve got a radical reduction of those and if the ones that are left are relatively dysfunctional, not even working if they work in at all, then you’ve got a problem.  So these cancer cells don’t have a choice, they have to revert to anaerobic metabolism.

Healthy mitochondria send signals, (epigenetic communication) between its cell and the nucleus and this epigenetic signalling from the mitochondria is actually what’s responsible for initiating a significant percentage of the genetic damage that has been identified so well through the DNA sequencing project, which the majority of the scientists have been focused on currently. 

If we’re ever going to cure cancer we have to focus on the metabolism.

Christofferson:  The mitochondrial damage is irrefutable. We look at cancer cells and the number of mitochondria are vastly reduced. When you isolate and look at the mitochondria you look at them, they’re messed up. There are protein problems; there are lipid problems, and all kinds of structural abnormalities. The question is: “Why has the cancer reverted to anaerobic energy generation?” Nobody really tied that to the terrible structure of the mitochondria. They didn’t have the tools to see the mitochondria and now we do.

Tom Seyfried, the leading edge cancer scientist, has done such a great job of piecing together the events once mitochondrial are damaged.  This relationship between mitochondria and the nucleus is so important. They constantly crosstalk and mitochondrial health is correlated to the health of the entire organism, you.

The dominant theory of aging explains that you age because your mitochondria age,they take the brunt of metabolism. When you generate energy you are whipping around free radicals and they’re constantly under stress so they get banged up and beat up and you look at the antioxidants within mitochondria they have declined by about 50% with advanced age.

Mercola: It is important to note, is when they burn fuel they generate these reactive oxygen systems (ROS). But the question is what you fuel are they’re burning?

 They are burning glucose (carbohydrates). They are burning dirty fuel generating tons of reactive oxygen species as opposed to burning ketones or fats.

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Christofferson:  As a fuel ketones are incredible, they burned clean with much less ROS or free radical generation but the clincher with this theory is that once there’s enough mitochondrial damage there is a retrograde response or an epigenetic signal to the nucleus and once this happens then you start to see the accumulation of mutations.

So the whole crux of this theory is which is first? The argument is that the metabolic theory is this mitochondrial damage is how first and then you see the mutations.The mutations appeared as the causebut in fact they are a downstream signalfrom the from the true cause. Researchers were led sort of down this wild-goose chase trying to find what these mutations were and why they were important. So now that we know this we can go back to the mitochondria and ask how do restore our cell and how do we keep them healthy?

The dietary therapy, what Tom Seyfried calls a restricted Ketogenic diet, is the foundation of this therapeutic approach because it does incredible things to the body where it differentiates between cancer cells and normal cells. When you switch from glucose metabolism to ketone metabolism you put energetic pressure on the cancer cells because they have to burn ketones in mitochondria, which is something they don’t have much of.

So we’ve noticed that once you put people in this dietary state everything becomes more effective even traditional chemotherapy even radiation and at the same time you’re mitigating side-effects because healthy tissues are able to withstand the sort of toxic payload from traditional chemotherapy. The exciting thing is when you add on these other metabolic therapies that are synergistic and their mechanisms overlap. Exogenous ketones are exciting.  How far can we take that metabolic state to this tipping point where cancer cells growth is stopped but they begin to die.

We can bring blood glucose levels down way farther than most people think safely. You add gluconeogenesis inhibitors that’s one of the main culprits you have to contend with as far as keeping blood glucose down, you add exogenously tones and people are put in this different metabolic state.

They’re shifting away from carbohydrate metabolism and then add on pulses of pressure: 3bromopyruvate(3-BrPA) and dichloroacetate (DCA) are inhibitors and Metformin. They are very non-toxic that overlap and mechanism.

This foundational shift to a ketone-based metabolism followed by these pulses of pressure appears to be the best avenue to a cure.  

Ketone bodies are not just a fuel they’re signalling molecules. Incredibly restorative things happen: you completely rearrange the architecture of your DNA; you start expressing new genes; you drop down the inflammatory genes and boost all these restorative genes. There is mitochondrial biogenesis. Look at all the diseases that spin out of this metabolic dysfunction from not entering the state of ketosis periodically – neurodegenerative diseases such as Alzheimer’s, Parkinson’s, obesity, diabetes, heart failure, heart disease, arthritis, cancer, and the list goes on.

A shift in macromolecular consumption from carbohydrates to fats can stop diseases. It goes back to Dr. Veech who was the lineage of Otto Warburg. He got his PhD in under Hans Krebs. He’s done a lot of this ketone body research along with Cahill and he said in late 70s or 80s that so many disease states spin out of a function of mitochondrial decay and the ketone bodies can potentially mitigate this process. Now we have exotic ketones that maybe help people that aren’t able to do the dietary restrictions.

The ketogenic diet really is just the maintenance of the fasting state nutritionally. Periodic fasting can be difficult because you transition through this induction phase. Your body is tooled up to burn carbohydrates, so when you fast it has to retool and during that process you burn muscle as a bridge to get you over to fat metabolism.

All evidence points to maybe not a huge increase in lifespan but it definitely a huge increase in health span. For example, if you were predisposed to getting type 2 diabetes in midlife, if you were in ketogenic state you may never get it. So if you want to live well for a long time that is were the benefit will be.

Here is the link to Travis Christofferson’s book Tripping over the Truth: How the Metabolic Theory of Cancer Is Overturning One of Medicine’s Most Entrenched Paradigms  https://www.amazon.ca/Tripping-over-Truth-Overturning-Entrenched/dp/1603587292

This Post has the highlights of the interview with Dr. Mercola & Travis Christofferson on the Metabolic Theory of Cancer https://www.youtube.com/watch?v=WvNaO0Poeqs

I invite you to Follow my Blog, Facebook or be added to my email distribution list. My focus is to maximize my physical performance and mental clarity, body composition, and most importantly overall health with a wholesome diet and exercise.

 I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

To follow my Blog, please click the Follow button to receive an email when the next posting is available. Hint: You may have to click the Accept and Close button before follow is available.

I thrive on feedback. Please let me know you are interested in the content by clicking Like, Commenting or sending me a message or email about the Post.

If you wish to contact me by Email, please email lpolstra@bell.net using this form.

May you Live Long Healthy.

Yours truly,

Lydia Polstra

Email: lpolstra@bell.net

Facebook: https://www.facebook.com/2healthyhabits/

Blog: https://2healthyhabits.wordpress.com

Disclaimer: The content of this email or Post is not intended for the treatment or prevention of disease, nor as a substitute for medical treatment, nor as an alternative to medical advice. Use of recommendations is at the choice and risk of the reader.

Diabetes – Three Reasons It’s Harder For People With Type 2 Diabetes To Lose Weight

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Approximately 90% of people with type 2 diabetes are overweight or obese.

While obesity often contributes to the development of diabetes, the bigger driver of weight gain is the high insulin levels that are found well before the diagnosis of diabetes.

There are some good reasons why the standard advice of “eat less, exercise more” doesn’t deliver results for people living with type 2 diabetes.

Reason #1: With type 2 diabetes, insulin is high, and insulin is a fat-storage hormone

Everyone has glucose, a type of sugar, in their blood at all times. Glucose is a source of energy that largely comes from eating carbohydrates. Simply put, when you eat carbohydrates, your blood sugar rises.

Insulin is produced by your pancreas, and insulin has many functions in the body. One of insulin’s functions is to help get glucose out of the blood and into cells where it can be used.  In order to do this, insulin rises along with glucose. So when you eat carbohydrates and glucose rises, the insulin is rising as well.

Once in the cells, glucose is mostly used for energy. If you have type 2 diabetes, this process doesn’t work well anymore: your body has become resistant to the signal of insulin, so the insulin isn’t as effective at moving the glucose out of your blood. That’s how you end up with high blood sugar levels after eating carbohydrates. Having chronically elevated blood sugar levels is dangerous, so your body needs to do something about it.

Your body responds by making more and more insulin to try to get the job done. Recall now that insulin has many functions, not just to facilitate the removal of glucose from the blood. Insulin also works to promote the storage of fat and to block the release of fat from fat storage. So instead of losing weight, you just keep gaining, thanks to all that insulin.

Reason #2: Typically recommended eating patterns often backfire by keeping you hungry and keeping your blood sugar high

If you’re like most people with type 2 diabetes, you’ve been told to eat carbohydrates but eat fewer overall calories, and to eat small meals throughout the day to keep your blood sugar steady; you’ve probably been advised to count your carbs and eat enough of them to keep your blood sugar up after taking medication to lower it—confusing, right?

What many find as a result is that they’re always hungry, always thinking about food and facing cravings. What’s at work is a survival instinct that even the strongest-willed person can’t withstand for long. This is a situation where your physiology is fighting against you. Even worse, those frequent small meals with carbohydrates create spikes in your blood sugar followed by drops in your blood sugar—a blood sugar roller-coaster that stimulates frequent hunger.

Reason #3: Type 2 diabetes medications can drive weight gain

Remember how your body’s own insulin is a fat-storage hormone? That’s also true for insulin that has been prescribed to you, whether delivered by injection or by pump. That’s why a common side effect of prescribed insulin is weight gain. Another class of medicine for type 2 diabetes, Sulfonylureas, work by stimulating the pancreas to produce more insulin. And once again, more insulin in your body means more fat storage and more weight gain.

The Accord Trial chart below shows that intensive insulin therapy caused significant weight gain.

Blog - Jan. 17 1. chart.

What’s the solution?

People living with type 2 diabetes are insulin resistant, meaning their tissues are not responding as they should to insulin. Insulin moves sugar from your blood into your cells. If your body does not respond to its own insulin, then your blood sugar will remain chronically elevated, and your body will produce more insulin. The most direct solution is to decrease the source of high blood sugar itself – carbohydrate consumption.

In fact, insulin resistance can be fundamentally referred to as “carbohydrate intolerance” because when carbohydrates are consumed by someone who is insulin resistant, blood glucose is not lowered as effectively. So, by eating fewer carbohydrates, we both reduce the glucose load in the blood, and decrease the release of insulin.

Nutritional ketosis is a natural metabolic state in which your body adapts to burning fat over carbohydrates as its primary fuel. While carbohydrate consumption triggers spikes in blood sugar, fat consumption does not, making it a better source of fuel for people with insulin resistance.

The graph below shows how carbs, protein and fat affect blood insulin levels.

Blog - Jan 17 2. carb, protein, fat

In a clinical trial, patients lost an average of 12% of their starting body weight within six months by using a medically supervised treatment that included the employment of nutritional ketosis.In addition, 56% of patients with type 2 diabetes reduced their HbA1c to below diabetic levels.

Read more about nutritional ketosis and how it can be an effective diabetes reversal method when paired with physician supervision here, Reversing Type 2 Diabetes  https://www.virtahealth.com/reversediabetes

To learn more about how food affects blood sugar, watch Sarah Hallberg, DO, MS on Carbohydrate Intolerance, Insulin Resistance and Reversing Diabetes. Here is the link https://www.youtube.com/watch?v=ldzaLP8oAHw&t=3s

The source of the Blog Post is found at the Virta website.  Please look for:3 Reasons It’s Harder For People With Type 2 Diabetes To Lose Weight. Please copy this link and paste in your address bar.

https://blog.virtahealth.com/3-reasons-its-harder-for-people-with-type-2-diabetes-to-lose-weight/?fbclid=IwAR0TKV7IDTDErccfXu27TfLFkDNojFoslpRqDFTff85W12gscf5AhYc255I

The citations show the supporting studies.

I invite you to Follow my Blog, Facebook or be added to my email distribution list. My focus is to maximize my physical performance and mental clarity, body composition, and most importantly overall health with a wholesome diet and exercise.

I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

To follow my Blog, please click the Follow button to receive an email when the next posting is available. Hint: You may have to click the Accept and Close button before follow is available.

I thrive on feedback. Please let me know you are interested in the content by clicking Like, Commenting or sending me a message or email about the Post.

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May you Live Long Healthy.

Yours truly,

Lydia Polstra

Facebook: https://www.facebook.com/2healthyhabits/

Blog: https://2healthyhabits.wordpress.com

Disclaimer: The content of this email or Post is not intended for the treatment or prevention of disease, nor as a substitute for medical treatment, nor as an alternative to medical advice. Use of recommendations is at the choice and risk of the reader.

OBESITY: Is it hiding a bigger problem? A doctor blamed his patient. Please listen to his tearful apology.

The following are the highlights of the video. I urge you to watch the video, the link is at the end of this Post.

Dr. Peter Attia says, I’ll never forget that day.

I was a surgical resident at The Johns Hopkins Hospital,taking emergency call. I got paged by the E.R. around 2 in the morning to come and see a woman with a diabetic ulcer on her foot.

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The question that was being asked of me was a did she also need an amputation?

I’d love so desperately to believe that I treated that woman on that night with empathy and compassion but I stood in the E.R. and determined that my diabetic patient did indeed need an amputation.

Why did I hold her in such bitter contempt? You see this woman had type 2 diabetes. She was fat.

And we all know that’s from eating too much and not exercising enough, right? I mean, how hard can it be?

As I looked down at her in the bed, I thought to myself, if you just tried caring even a little bit, you wouldn’t be in this situation at this moment with some doctor you’ve never met about to amputate your foot.

Why did I feel justified in judging her? Because I thought I had her all figured out. She ate too much. She got diabetes. Case closed.

I never once questioned the conventional wisdom.

Three years later, I found out how wrong I was.

But this time, I was the patient.

Despite exercising three or four hours every single day, and following the food pyramid to the letter, I’d gained a lot of weight and developed something called metabolic syndrome.

I had become insulin-resistant. You can think of insulin as this master hormone that controls what our body does with the foods we eat, whether we burn it or store it, (fuel partitioning).

Now failure to produce enough insulin is incompatible with life. Insulin resistance is when your cells get increasingly resistant to the effect of insulin trying to do its job.

Once you’re insulin-resistant, you’re on your way to getting diabetes, which is what happens when your pancreas can’t keep up with the resistance and make enough insulin.

Now your blood sugar levels start to rise, which can lead to heart disease, cancer, even Alzheimer’s disease, and amputations.

With that scare, I got busy changing my diet. I lost 40 pounds, weirdly while exercising less. I’m not overweight anymore. I don’t have insulin resistance.

Most researchers believe obesity is the cause of insulin resistance. If you want to treat insulin resistance, you get people to lose weight, right?

But what if we have it backwards? What if obesity is a coping mechanism for a far more sinister problem going on underneath the cell?

Insulin resistance is the reduced capacity of our cells to partition fuel.  When we become insulin-resistant, the insulin says to a cell, I want you to burn more energy than the cell considers safe, the cell, in effect, says,

“No thanks, I’d actually rather store this energy.”

And because fat cells are actually missing most of the complex cellular machinery found in other cells, it’s probably the safest place to store it. We become fat.

This is a really subtle distinction. Consider the following analogy: Think of the bruise you get on your shin when you inadvertently bang your leg into the coffee table. Sure, the bruise hurts like hell, and you almost certainly don’t like the discolored look, but the bruise is not the problem.

In fact, it’s the opposite. It’s a healthy response to the trauma, all of those immune cells rushing to the site of the injury to salvage cellular debris and prevent the spread of infection to elsewhere in the body.

Now, imagine we thought bruises were the problem, and we evolved a giant medical establishment and a culture around treating bruises: masking creams, painkillers, etc., all the while ignoring the fact that people are still banging their shins into coffee tables.

How much better would we be if we treated the cause – move the coffee table so people don’t injure themselves – rather than the effect?

Getting the cause and the effect right makes all the difference in the world. Getting it wrong, and the pharmaceutical industry can still do very well for its shareholders but nothing improves for the people with bruised shins.  Cause and effect.

We have the cause and effect wrong on obesity and insulin resistance. Maybe we should be asking ourselves, is it possible that insulin resistance causes weight gain and the diseases associated with obesity, at least in most people?

What if being obese is just a metabolic response to something much more threatening, an underlying epidemic?

So what if we’re fighting the wrong war, fighting obesity rather than insulin resistance?

Even worse, what if blaming the obese means we’re blaming the victims?

What if some of our fundamental ideas about obesity are just wrong?

What is a cell is trying to protect itself from when it becomes insulin resistant, the answer probably isn’t too much food. It’s more likely too much glucose: blood sugar.

Now, we know that refined grains and starches elevate your blood sugar in the short run, and there’s even reason to believe that sugar may lead to insulin resistance directly.

It might be our increased intake of refined grains, sugars and starches that’s driving this epidemic of obesity and diabetes, but through insulin resistance.

It is not necessarily through just overeating and under-exercising.

When I lost my 40 pounds a few years ago, I did it simply by restricting those things.

We can’t keep blaming our overweight and diabetic patients like I did.

Most of them actually want to do the right thing, but they have to know what that is, and it’s got to work.

One day people will shed their excess pounds and cure themselves of insulin resistance.

Sometimes I think back to that night in the E.R., I wish I could speak with that woman again.

I’d like to tell her how sorry I am.

You didn’t need my judgment and my contempt. You needed my empathy and compassion.

I hope you can forgive me.

Please watch the moving video: Peter Attia: What if we’re wrong about diabetes?

Next Week the Post will be from 3 Reasons It’s Harder For People With Type 2 Diabetes To Lose Weight

https://blog.virtahealth.com/3-reasons-its-harder-for-people-with-type-2-diabetes-to-lose-weight/?fbclid=IwAR0TKV7IDTDErccfXu27TfLFkDNojFoslpRqDFTff85W12gscf5AhYc255I

I invite you to Follow my Blog, Facebook or be added to my email distribution list. My focus is to maximize my physical performance and mental clarity, body composition, and most importantly overall health with a wholesome diet and exercise.

 I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

To follow my Blog, please click the Follow button to receive an email when the next posting is available. Hint: You may have to click the Accept and Close button before follow is available.

 I thrive on feedback. Please let me know you are interested in the content by clicking Like, Commenting or sending me a message or email about the Post.

If you wish to contact me by Email, please email lpolstra@bell.net using this form.

May you Live Long Healthy.

Yours truly,

Lydia Polstra

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Blog: https://2healthyhabits.wordpress.com

Disclaimer: The content of this email or Post is not intended for the treatment or prevention of disease, nor as a substitute for medical treatment, nor as an alternative to medical advice. Use of recommendations is at the choice and risk of the reader.

Guide To Making Keto Work Long Term

  1. Add fat to your foods so you aren’t hungry, but only until you are satisfied, adding too much fat will stall your weight loss. Keep a staple like spreadable butter blend on hand and customize it with your favorite herbs or flavors (like garlic!)
Blog - 1. Fats

Learn more at Which Fats Do I Use To Replace Vegetable Oils?

Which Fats Do I Use To Replace Vegetable Oils?

  1. Substitute ‘must-haves’ and treats with a low-carb option.

These recipes and more can be found at Bounti https://buttoni.wordpress.com/

Blog - 2 cake

Maple Walnut Ice Cream

Pecan Bundt Cake

Toscana Soup

Nachos

  1. Keto doesn’t need to be meat heavy. Focus on carbohydrate restriction and eat foods that appeal to you in order to make this work long term.
Blog - 3 veg

How much? Learn more at How Much Protein Do You Need In Nutritional Ketosis?

How Much Protein Do You Need In Nutritional Ketosis?

  1. Try new foods and recipes to avoid boredom.For example, this low carb bread recipehttps://www.ketoconnect.net/best-keto-bread/#wprm-recipe-container-16189
Blog - 4 bread
  1. Pack snacks for travel or emergencies.See our travel and on-the-go tips.
Blog - 5 Snacks on the go

Mario Olives

Macadamia Nuts

Low-carb meat sticks

Moon Cheese

Pork Rinds

  1. Take low-carb dishes to parties and gatherings that no one will realize are low carb. These and other Keto recipes can be found at https://www.ketoconnect.net/
Blog - 6 mashed caulifour

Green Bean Casserole

Mashed Cauliflower

Dinner Rolls

For more information please go to How to Travel While Staying Keto/ Low Carb.  https://2healthyhabits.wordpress.com/?s=How+to+Travel+While+Staying+Keto%2F+Low+Carb

  1. Hydrate smart.You will need to make sure you’re drinking enough water and adding electrolytes like sodium to avoid some common side effects.
Blog - 7 Water glass

* If you have high blood pressure lowering medication or on blood sugar lowering medications, make sure you check with a doctor before staring low carb or keto.  Consume at least 64 ounces of fluids daily.

Blog - 7a - Salt

Schedule two bouillon into your daily routine (unless otherwise prescribed) and salt your foods to taste.

How much salt (sodium) do you need?  Learn more at Managing Potassium and Sodium On a Ketogenic Diet

Managing Potassium and Sodium On a Ketogenic Diet

  1. You don’t have to stop drinking alcohol but cap it at two of these lower carb alcohols.
Blog - 8 alcohol

Drinking can make sticking to healthy habits more difficult in many ways. For one, the extra carbohydrates and calories in alcohol add up. Further, drinking lowers inhibitions – which may make you more likely to eat off plan.

If you know you are going to drink alcohol, stick to lower carbohydrate options and consider using seltzer to cut drinks.

Did you know that alcohol, depending on the amount, is burned as fuel before MCT oil, ketones or body fat and take you out of ketosis?

Remember why you started your diet and how hard it was to get into this healthy state, that may help you resist temptation.

Do you have type 2 diabetes and want to lose weight?

Several studies have reported successful weight loss with decreased insulin resistance and medication use following a Low Carb Diet. Research suggests that Low Carb Diet’s are effective in reversing diabetes in the short term (up to two years), especially in patients with a more recent diabetes diagnosis.

To learn more read Reversing Type 2 Diabetes with Bariatric Surgery, Very Low Calorie Diets, and Carbohydrate Restriction: A Review of the Evidence

Reversing Type 2 Diabetes with Bariatric Surgery, Very Low Calorie Diets, and Carbohydrate Restriction: A Review of the Evidence

The source of this post is Guide To Making Keto Work

https://about.virtahealth.com/virta-guide-to-making-keto-work?_ga=2.137694835.2122312368.1569418254-1797598660.1569418254

Learn more about staying on the Ketogenic diet long term at https://2healthyhabits.wordpress.com/2017/04/14/ketosis-dr-phinney-tells-us-to-maintain-a-low-carb-diet-for-a-long-time/

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I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

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May you Live Long Healthy.

Yours truly,

Lydia Polstra

Facebook: https://www.facebook.com/2healthyhabits/

Blog: https://2healthyhabits.wordpress.com

Disclaimer: The content of this email or Post is not intended for the treatment or prevention of disease, nor as a substitute for medical treatment, nor as an alternative to medical advice. Use of recommendations is at the choice and risk of the reader.