Statins – 2HealthyHabits

Saturated Fat Part Two: Gauging Your Heart Disease Risk

You are at risk of a heart disease if you have 3 out of the following 5 indications of metabolic syndrome:

A fasting insulin level above 3

A triglyceride to HDL ratio above 2

Low HDL

A waist circumference indicating overweight or obesity

High blood pressure

Another major risk factor for heart disease that receives virtually no attention is high iron levels. In menstruating women, this is not an issue since they lose blood on a monthly basis. This is actually part of why premenopausal women have a decreased risk of heart disease.

In men, iron levels can rise to dangerously high levels. In my experience, the majority of adult males and postmenopausal women have elevated levels that put their health at risk. Checking your iron levels is easy and can be done with a simple blood test called a serum ferritin test. If your levels are high, all you have to do is donate blood a few times a year.

The Connection Between Saturated Fats and Diabetes

Malhotra cites a 2014 Lancet study looking at the association between dietary saturated fat, plasma saturated fat and type 2 diabetes. Interestingly, while dietary saturated fats found in dairy products were strongly inversely associated with the development of type 2 diabetes (meaning it was protective), endogenously-synthesized plasma-saturated fat was strongly associated with an increased risk.

Endogenously-synthesized plasma-saturated fats are fatty acids produced by your liver in response to net carbohydrates, sugar and alcohol.These findings suggest eating full-fat dairy products may protect you against type 2 diabetes,whereas consuming too many net carbs (total carbs minus fiber) will increase your risk of type 2 diabetes –in part by raising the saturated fat levels in your bloodstream.

Healthy Fat Tips

Here are a few tips to help ensure you’re eating the right fats for your health:

Use organic butter made from raw grass-fed milk instead of margarines and vegetable oil spreads.
Use coconut oil for cooking. It is primarily a saturated fat and more resistant to heat damage than other cooking oils. It will also help improve your ability to burn fat and serve as a great source of energy to help you make the transition to burning fat for fuel.
Sardines and anchovies are an excellent source of beneficial omega-3 fats and are also very low in toxins that are present in most other fish.
To round out your healthy fat intake, be sure to eat raw fats, such as those from avocados, raw dairy products, and olive oil, and also take a high-quality source of animal-based omega-3 fat, such as krill oil.

Why Statins Are a Bad Idea for Most People

In addition to the recommendation to follow a low-fat diet, many doctors are still avid prescribers of statins, which help lower your cholesterol.

“This is a drug that was marketed over the last three decades as being a wonder drug. We’re only now realizing that the benefits of statins have been grossly exaggerated and the side effects underplayed. One of the reasons for that is that most if not all of the studies that drove the guidelines were industry-sponsored studies.

One of the things we have neglected in medicine is this issue around absolute risk and relative risk. The reality is if you look at the published data … if you have heart disease and you’ve had a heart attack, then taking a statin every day for five years, there’s a 1 in 83 chance that [statin] will save your life.

That means in 82 of 83 cases, it’s not going to save your life.

People with low risk should know that if they haven’t had a heart attack, according to the published literature, they’re going to live one day longer from taking statins.”

Statins Are Associated With Serious Side Effects

Then there’s the issue of side effects. According to Malhotra, between 1 in 3 and 1 in 5 patients suffer unacceptable side effects (that interfere with or diminish the quality of your life). Muscle pain is the most significant side effect reported followed by fatigue (mostly in women) because statins are essentially a metabolic blocker and mitochondrial poison.

They inhibit an enzyme called HMG-CoA reductase. This is how they lower cholesterol. But that same enzyme is also responsible for a number of other things like making coenzyme Q10, which is why muscle pain and fatigue are so common. This is in fact a sign that your CoQ10 is being depleted, and you don’t have enough cellular energy.

Statins also block the formation of ketones, which are an essential part of mitochondrial nutrition and overall health. If you can’t make ketones, you impair the metabolism in your entire body, including your heart, thereby raising your risk for heart problems and a variety of other diseases. It’s also recently been established that within a few years of taking statins, the drug causes type 2 diabetes in one out of 100 patients.

That too can be a significant tradeoff that needs to be taken into account, as diabetes is a risk factor for heart disease and other chronic diseases. Dr. Michel De Lorgeril, a well-respected French cardiologist at Grenoble University recently reopened the debate about statins after publishing a review in which he questions whether statins actually have any benefit at all.

“He pointed out several discrepancies in the original trials … statistical manipulation, conflict of interest … ” Malhotra says. ”He’s actually suggested that maybe nobody benefits from statins; even people on statins for prevention.

He says that unless we get access to the raw data, independent analysis, the actual claims about the benefits of statins are not evidence-based.”

In case you missed it, please read

Saturated Fat Part One: Great Britain’s Most Outspoken Cardiologist Sets the Record Straight

Is saturated fat dangerous to your health?

Hospitals and Medical Personnel Are Far From Paragons of Health

For Past 60 Years, the Wrong Fats Have Been Vilified

What Are the Real Risk Factors for Heart Disease?

Here is the link https://2healthyhabits.wordpress.com/2020/02/28/saturated-fats-part-one-great-britains-most-outspoken-cardiologist-sets-the-record-straight/

Part One and Two have been condensed from Dr. Mercola’s post: Great Britain’s Most Outspoken Cardiologist Sets the Record Straight on Saturated Fats

https://articles.mercola.com/sites/articles/archive/2016/06/05/saturated-fat-heart-disease-risk.aspx

Please see the original for the Footnotes and Citations for the scientific studies.

May you Live Long Healthy.

Yours truly,

Lydia Polstra

Email: lpolstra@sympatico.ca

Facebook: https://www.facebook.com/2healthyhabits/

Blog: https://2healthyhabits.wordpress.com

Disclaimer: The content of this email or Post is not intended for the treatment or prevention of disease, nor as a substitute for medical treatment, nor as an alternative to medical advice. Use of recommendations is at the choice and risk of the reader.

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World Renowned Heart Surgeon Speaks Out On What Really Causes Heart Disease.

Dr. Dwight Lundell is the past Chief of Staff and Chief of Surgery at Banner Heart Hospital, Mesa , AZ. His private practice, Cardiac Care Center was in Mesa, AZ. Recently Dr. Lundell left surgery to focus on the nutritional treatment of heart disease. He is the founder of Healthy Humans Foundation that promotes human health with a focus on helping large corporations promote wellness. He is also the author of The Cure for Heart Disease and The Great Cholesterol Lie.

Dr. Lundell says, as a heart surgeon with 25 years experience, having performed over 5,000 open-heart surgeries, today is my day to right the wrong with medical and scientific fact. I trained for many years with other prominent physicians labeled “opinion makers.” Bombarded with scientific literature, continually attending education seminars, we opinion makers insisted heart disease resulted from the simple fact of elevated blood cholesterol. The only accepted therapy was prescribing medications to lower cholesterol and a diet that severely restricted fat intake. The latter of course we insisted would lower cholesterol and heart disease.

Deviations from these recommendations were considered heresy and could quite possibly result in malpractice.

What follows are the highlights from the video:

World Renowned Heart Surgeon Speaks Out On What Really Causes Heart Disease.

Dr. Lundell observed while doing heart operations that the cornonary artery had a lot of redness and swelling around the plaque area. These are two of the cardinal signs of inflammation.

He began wondering if inflammation was part of the problem. Russell Ross and others published and articles about vascular biology, that proved inflammation was the mechanism behind plaque build up in the arteries.

The cause is not cholesterol. 70% of heart attack patients have normal cholesterol.

Scientific studies after 2006 had more stringent guides and showed that statins do not reduce the risk of heart attack.

Here is a video of one person’s experience with statins World Renowned Heart Surgeon Speaks Out On What Really Causes Heart Disease

Dr. Lundell says that cholesterol is not a marker for heart disease. It is a marker for eating too many carbohydrates because carbohydrate gets turned into triglycerides, which raises your LDL cholesterol. To lower your cholesterol, eat a lot of saturated fats and lower your carbohydrate intake.

Cholesterol is not important. What is important is sugar, that’s carbohydrate.

The 1977 US food guide recommended 60 to 70% of the food intake should be carbohydrates and eliminated saturated fats.

Saturated fat raises LDL cholesterol. If LDL cholesterol is not the cause of heart disease, eliminating saturated that makes no sense.

What is the real cause of heart disease? It is inflammation. Inflammation follows injury. What is causing the injury? It is sugar.

Dr. Michael Brownlee published an article detailing the mechanisms by which sugar damages. The cells in the eyes and the kidneys are different from the rest of the body. They cannot stop sugar or glucose going into their cells so they were damaged more quickly than other cells. Dr. Brownlee wanted to know how these cells were so injured as to cause blindness and kidney disease. He found that injury causes inflammation, and inflammation is the mechanism for heart disease as well as other diseases. But what’s causing the injury?

Sugar is causing it. Sugar molecules combined with protein or fat in a process called glycation. A1c is glycated hemoglobin. The main pathway to inflammation is when the sugar (glucose) gets presented to the mitochondria inside endothelial cell and since it can’t stop sugar coming in it gets damaged. (Endothelial cells normally line blood vessels to maintain vascular integrity and permeability).

When sugar is introduced to the mitochondria it overloads it and produces a whole bunch of extra free radicals, which then caused damages to the cell, which then trigger the inflammation. Plaque as produced as a bandage over the inflammation.

The standard American diet injures the cells every day. It is the main cause of heart disease. Not cholesterol, not salt. It’s sugar.

Other cells can stop glucose sugar from coming in that is the essence of insulin resistance causing diabetes.

If you want to be healthy and control blood sugar, stop eating a standard American diet. Get yourself on a reduced low carbohydrate diet with extra healthy saturated fat and a moderate amount of protein.

Low carbohydrate nutrition is the key to health.

I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

To follow my Blog, please click the Follow button to receive an email when the next posting is available. Hint: You may have to click the Accept and Close button before follow is available.

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May you Live Long Healthy.

Yours truly,

Lydia Polstra

Email: lpolstra@bell.net

Facebook: https://www.facebook.com/2healthyhabits/

Blog: https://2healthyhabits.wordpress.com

Cholesterol: The Standard Measure Of Blood LDL Cholesterol Is Inaccurate

For the last 5 decades, most medical and nutrition scientists have focused on low-density lipoprotein (LDL) cholesterol (“bad cholesterol”) as a primary cause of coronary heart disease. While cholesterol lowering therapy has become the standard of care for some individuals with well-defined heart disease risk, this focus on cholesterol in general – and LDL cholesterol in particular – remains very controversial.

Unfortunately the standard measure of blood LDL cholesterol is inaccurate, and when the various components of the blood LDL are measured they represent only a fraction of the lipid (fatty acids) and other biomarkers of heart disease risk.

A turning point in understanding the limitations of LDL and heart disease came with the publication of the Lyon Diet Heart Study. This randomized trial pitted a standard low fat diet against a Mediterranean diet for people with a prior heart attack. There was no difference in LDL cholesterol changes between the Mediterranean and low fat diet groups. The standard calculated LDL value did not seem to matter that much indicating that some very important drivers of coronary disease risk were going unmeasured.

In the recently published 1-year results from the IUH/Virta diabetes reversal study, they reported a small but significant rise in the average blood LDL cholesterol level in patients on a well-formulated ketogenic diet (WFKD). At the same time, however, they noted major reductions in a number of coronary disease risk factors including weight, blood pressure, and HbA1c.

The pattern of how these other risk factors change is independent of the changes in LDL.

In Figure 1, (please see the original post) the number of patients whose LDL went up was somewhat greater than the number whose LDL went down.

In the Figure 2, the 14 other risk factors with improved risk far outweigh those indicating a negative response. The heart disease risk factors indicate that a WFKD is much healthier than one might conclude by focusing on the single LDL risk factor.

Novel results from the IUH Diabetes Reversal Study:

Figure 2

Blog - July 26 actual numbers for each of the risk factors -3.png

Here is a table (figure 2) with the actual numbers for each of the risk factors shown in figure 1, plus HbA1c and weight. For each factor, there is a change between baseline and 1-year as a percent, the P-value indicating the chance this change might occur at random (i.e., the smaller the better), and then arrows up or down indicating how this change might influence overall heart disease and mortality risk.

These results are interesting for two reasons. First, none of the short term studies of ketogenic diets given to patients with type 2 diabetes lasting a few week to a few months have sustained these benefits without weight regain and/or rising HbA1c values after 1 year.

Second, no prior prospective outpatient low carbohydrate diet study has documented anything near this degree (0.6 mM at 10 weeks and 0.3 to 0.4 mM at one year) of sustained nutritional ketosis in such a large group past the first few months of treatment.

Note that only the arrow for calculated LDL cholesterol points up, whereas all of the other 16 point down. Also note that of these 16, only two have associated P-values that are non-significant or borderline. All of the others indicate a chance of random error of less than 1-in-ten-thousand. This means in turn is that these observations of reduced risk are statistically very strong.

This sustained nutritional ketosis may be a pivotal factor in risk reductions. The resultant beta-hydroxybutyrate (BOHB) is now understood to be a potent epigenetic signal controlling the body’s defenses against oxidative stress, inflammation, and insulin resistance.

What Blood Lipids Actually Do:

We have known for a long time that blood levels of both cholesterol and saturated fat tend to be independent of how much of these nutrients we eat.

But a completely different set of factors come into play when we eat less energy than we burn, which forces the body to dip into its energy stores and thus mobilize body fat. Similarly, when we restrict dietary carbohydrates, either body fat or dietary fat has to become the body’s principle source of energy. This is most profound in the keto-adapted state, where circulating lipids and blood ketones (made from fat in the liver) together provide 75-85% of the body’s energy. Lipoproteins play a critical role in the transport of lipid in the bloodstream, so changes in delivery needs in turn will impact lipoprotein lab results.

In response to the state of nutritional ketosis, humans can more than double their rates of fat oxidation (i.e., use for fuel) at rest and during exercise. So when you combine a doubling of fat intake with a doubling of fat use, clearly a lot more fat has to pass through the bloodstream.

We have spent a few decades studying how the body adapts to a WFKD, and have come to recognize a pretty consistent pattern for the changes in most blood lipids, but a quite inconsistent pattern for the calculated LDL cholesterol level in particular. The consistent changes are:

A dramatic reduction in serum triglycerides
A rise in HDL cholesterol and
The same or lower levels of saturated fats in serum triglycerides

The one inconsistent variable in this otherwise predictable pattern of change is the calculated serum LDL cholesterol level. For some people, following a ketogenic diet makes their LDL cholesterol go down and for some it does not change. But for a fair number of people the calculated LDL cholesterol value rises, in some cases quite a lot. Thus the key question is: How important is the calculated LDL cholesterol relative to the other risk factors that have been recently characterized?

Why LDL Cholesterol is Not a Single Number:

There are two important limitations of the commonly reported serum LDL cholesterol level.

First, the usual test procedure does not actually measure LDL – it reports a calculated value based upon measurements of serum total and HDL cholesterol and triglycerides, along with a number of assumptions. And in particular, when the triglyceride value undergoes a big change, it can skew the calculated LDL value considerably.

Second, the circulating lipoprotein particles classed as LDL are actually quite diverse in size, and it is now recognized that the smaller, more dense particles (which carry proportionately less triglyceride) are the sub-fraction that is associated with vascular damage and heart disease.

In Figure 2, only the calculated LDL cholesterol value went in the ‘wrong direction.’ But in this case, it appears that this was due to a shift to a greater proportion of the larger particles because the total particle number did not change, the average particle size increased, while the small dense particle number was significantly decreased.

Inflammation as an Independent Risk Factor for Coronary Vascular Disease:

Three decades ago, a number of mainstream investigators noted that the total white blood cell count and then c-reactive protein levels appear to predict coronary disease and mortality independent of cholesterol.

The key question as to whether this was a mere association or causal. This question was resolved with the recent CANTOS Trial, which reduced coronary risk by 15% without any effect on LDL cholesterol levels.

Unfortunately a side effect of the antibody used in this study (an increase in fatal infections) cancelled out the coronary disease risk reduction, so the CANTOS Trial answered an important question but did not offer a therapeutic solution. However this does offer an interesting insight into the mystery of why the Lyon Diet Heart Study reduced coronary disease risk AND mortality. In that study, blood anti-oxidant levels increased and granulocytes (aka white blood cells) decreased, suggesting that a dietary anti-inflammatory intervention can have potent benefits absent the dangerous side-effects of anti-inflammatory drugs.

Blood Saturated Fats and CVD Risk:

The key fact to keep in mind is that while a primary source of saturated fat in the blood is de novo lipogenesis (i.e., the production of fat from excess dietary carbohydrate), there is little if any relationship between dietary saturated fat intake and blood saturated fat content. And most importantly, because the keto-adapted state doubles the body’s ability to burn saturated fat for fuel while at the same time shutting down de novo lipogenesis, blood levels of saturated fats are reduced independent of dietary saturated fat intake.

There is a review of this topic in the blog, The Sad Saga of Saturated Fat. https://wordpress.com/post/2healthyhabits.wordpress.com/698

Getting Beyond Single Biomarkers of Cardiovascular Risk:

There is a wide range of processes that contribute to atherosclerosis and coronary artery disease risk. American College of Cardiology offers a 10-year heart disease risk predictor that includes the following inputs: age, sex, race, total cholesterol, HDL cholesterol, LDL cholesterol, systolic blood pressure, diastolic blood pressure, diabetes, smoking and medication use (statin, hypertension, aspirin).

Based upon this equation, the 10-year atherosclerotic cardiovascular disease (ASCVD) risk score in the recent Virta study decreased -11.9% (P = 5×10-5). This is a large beneficial effect experienced by the participants in the Virta study despite the observed changes in calculated LDL values.

Continuous doctor’s care treatment including nutritional ketosis in patients with type 2 diabetes improved most biomarkers of CVD risk after the Virta one-year study. The increase in LDL-cholesterol appeared limited to the large LDL sub-fraction; whereas LDL particle size increased, total LDL-P and ApoB were unchanged, and inflammation and blood pressure decreased.

There so many different factors are associated with coronary risk we need to avoid focusing on just LDL.

This Post has been condensed from the Virta blog: Blood Lipid Changes With A Well-Formulated Ketogenic Diet In Context by Rich Wood, PhD, Amy McKenzie, PhD, Jeff Volek, PhD, RD, Stephen Phinney, MD, PhD on May 2, 2018. Please see the original for the Footnotes and Citations for the scientific studies. Here is the link https://blog.virtahealth.com/blood-lipid-changes-with-ketogenic-diet/

I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

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Inflammation, Nutritional Ketosis, Type 2 Diabetes and Keto-Immune Modulation

Nutritional ketosis has anti-inflammatory and immune-modulating effects that are as potent as the most powerful drugs. This explains how a well-formulated ketogenic diet reverses type 2 diabetes, pre-diabetes, and metabolic syndrome.

Inflammation enables our bodies to recognize and respond to infection and injury. Having too weak of an inflammatory response leaves us prone to infection or impaired healing. But having too great of a response, or one that remains over-active for too long, puts us at risk for a form of chronic injury that underlies type 2 diabetes, coronary heart disease, many common cancers, and Alzheimer’s disease.

This balance between too little and too much inflammation is regulated by a number of circumstances including our genetic inheritance, toxins in the environment, and by many components of our diet.

Currently, we have a host of different drug classes designed to modulate inflammation, but safely managing their dose and duration of use requires professional vigilance to avoid dangerous side effects.

In the past decade, nutritional ketosis has emerged as a potent modulator of inflammation. And, unlike drugs that typically target just one aspect of the body’s immune response, keto-immuno-modulation (KIM) seems to work evenly to balance the anti-inflammatory effect in a safe, sustainable and surprisingly potent way without the serious side effects that characterize most pharmaceuticals.

Measuring Inflammation Levels

High-normal white blood cell count (WBC) levels as well as another test reflective of inflammation in the body called C-reactive protein (CRP) have been shown to also predict the development of type 2 diabetes, many common forms of cancer, and probably Alzheimer’s.

Complexity of Inflammation at a Glance

When there is too much inflammation a class of disorders called auto-immune disease can occur and the body’s activated defenses attack some of its own organs, causing conditions like rheumatoid arthritis, lupus, psoriasis, and type 1 diabetes. These immune disorders can result in an increased risk of heart disease, as are people with type 2 diabetes.

Drugs that Reduce Inflammation

Older, established drugs, like aspirin, tend to have more general modes of action and a broader spectrum of side effects. Recent pharmaceutical research has moved to target specific enzymes, bioactive molecules, or white blood cell types involved in inflammation to try to reduce side effects, but by focusing on just one single step in the complex cascade of the inflammation/immune system, there is a strong tendency to distort this system rather than reduce the inflammatory effect in a balanced manner.

The risks associated with chronic use of a variety of anti-inflammatory drugs often outweigh the desired benefits.

One example, when aspirin is used routinely in people without known heart disease (primary prevention), fatal hemorrhage is significantly increased.

Dietary Anti-inflammatory Treatments

A natural form of vitamin E – gamma-tocopherol (rather than alpha-tocopherol) has potent anti-inflammatory and oxidative stress lowering properties when used alone or in combination with the omega-3 fatty acid DHA.

Weight loss itself has been shown to reduce inflammation, and it appears that the greater the weight loss the larger the anti-inflammatory effect. This could be attributable to a reduction in the amount of very inflammatory belly fat, and/or a result of some patients being in nutritional ketosis.

Beta-hydroxybutyrate has potent regulatory effects on inflammation

Among the many ‘nutritional factors’ with potential anti-inflammatory properties, the ketone beta-hydroxybutyrate (BOHB) is emerging as both highly potent and uniquely safe as a long-term treatment for inflammation. When in the physiologically normal range that is seen with nutritional ketosis, BOHB activates a number of different genes that protect our cells from oxidative stress and inflammation.

Reactive oxygen species (ROS), aka ‘free radicals’ appear to be connected to inflammation. NSAIDs can’t block them. BOHB prevents this whole class of pro-inflammatory compounds from being created in the first place.

Please see the original Virta Post for the diagramof the contrasting effects of diets containing carbohydrate-plus-protein totals above 30% (thus suppressing ketogenesis) and a ketogenic diet on down-stream inflammatory pathways regulated by BOHB.

Upon starting a well-formulated ketogenic diet, the fatty acid most commonly attacked by ROS, called arachidonic acid, promptly increases. Much less AA is being destroyed by ROS when the body is in nutritional ketosis, therefore less needs to be made in order to maintain optimum membrane levels of this important essential fatty acid.

The level of AA in muscle membrane is strongly correlated with insulin sensitivity thus offering an explanation for the prompt improvement in insulin sensitivity upon initiation of a ketogenic diet.

Clinical Studies Demonstrating Reduced Inflammation

In a randomized trial comparing two weight loss diets – one ketogenic and the other low fat, high carbohydrate – the ketogenic diet demonstrated much greater anti-inflammatory effects after 12 weeks. Additionally, in our Virta/IUH study of patients with type 2 diabetes, both WBC count and C-reactive protein (CRP) were dramatically reduced in the ketogenic diet group compared to the usual care group at 1 and 2-year follow-up. In particular, the reduction in CRP in the ketogenic diet group at 1 year was comparable in magnitude (35-40%) to what is seen with the most potent statin drug.But unlike the statin, which appears to be primarily focused on CRP and has no effect on WBC count, nutritional ketosis addresses both, providing a more balanced effect on the network of interacting bioactive components influencing inflammation.

Perhaps, nutritional ketosis should be considered the new metabolic normal for people with diseases associated with or caused by chronic inflammation.

This Post has been condensed from Inflammation, Nutritional Ketosis, Type 2 Diabetes and Keto-Immune Modulation by Stephen Phinney, MD, PhD, Bailey, Ph.D., Jeff Volek, PhD, RD January 3, 2019 https://blog.virtahealth.com/ketone-supplements/https://blog.virtahealth.com/inflammation-ketosis-diabetes/More scientific information and Citations of the supporting studies are included in the Virta post.

Not sure what the Ketogenic diet is? Please read, What is the Ketogenic Diet? , in my Blog https://2healthyhabits.wordpress.com/2018/02/02/what-is-the-ketogenic-diet/

I will bring you compelling articles on Ketogenic and GAPS diets, the Super Slow High-Intensity Exercise Program and supplements.

To follow my Blog, please click the Follow button to receive an email when the next posting is available. Hint: You may have to click the Accept and Close button before follow is available.

I thrive on feedback. Please let me know you are interested in the content by clicking Like, Commenting or sending me a message or email about the Post.

If you wish to contact me by Email, please email lpolstra@bell.net using this form.

May you Live Long Healthy.

Yours truly,

Lydia Polstra

Email: lpolstra@bell.net

Facebook: https://www.facebook.com/2healthyhabits/

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